EMBL Heidelberg, Meyerhofstraße 1, 69117 Heidelberg, Germany
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Total: 13 publication(s)
Kultima JR, Coelho LP, Forslund K, Huerta-Cepas J, Li SS, Driessen M, Voigt AY, Zeller G, Sunagawa S, Bork P. (2016)
MOCAT2: a metagenomic assembly, annotation and profiling framework.
Voigt AY, Costea PI, Kultima JR, Li SS, Zeller G, Sunagawa S, Bork P. (2015)
Temporal and technical variability of human gut metagenomes.
Schramm SJ, Jayaswal V, Goel A, Li SS, Yang YH, Mann GJ, Wilkins MR. (2013)
Molecular interaction networks for the analysis of human disease: utility, limitations, and considerations.
Schramm SJ, Li SS, Jayaswal V, Fung DC, Campain AE, Pang CN, Scolyer RA, Yang YH, Mann GJ, Wilkins MR. (2013)
Disturbed protein-protein interaction networks in metastatic melanoma are associated with worse prognosis and increased functional mutation burden.
Pang CN, Goel A, Li SS, Wilkins MR. (2012)
A multidimensional matrix for systems biology research and its application to interaction networks.
Fung DC, Li SS, Goel A, Hong SH, Wilkins MR. (2012)
Visualization of the interactome: what are we looking at?
Ayer A, Fellermeier S, Fife C, Li SS, Smits G, Meyer AJ, Dawes IW, Perrone GG. (2011)
A genome-wide screen in yeast identifies specific oxidative stress genes required for the maintenance of sub-cellular redox homeostasis.
Chong HS, Campbell L, Padula MP, Hill C, Harry E, Li SS, Wilkins MR, Herbert B, Carter D. (2011)
Time-course proteome analysis reveals the dynamic response of Cryptococcus gattii cells to fluconazole.
Li SS, Xu K, Wilkins MR. (2011)
Visualization and analysis of the complexome network of Saccharomyces cerevisiae.
Goel A, Li SS, Wilkins MR. (2011)
Four-dimensional visualisation and analysis of protein-protein interaction networks.
Deshpande NP, Kaakoush NO, Mitchell H, Janitz K, Raftery MJ, Li SS, Wilkins MR. (2010)
Sequencing and validation of the genome of a Campylobacter concisus reveals intra-species diversity.
Ma L, Pang CN, Li SS, Wilkins MR. (2010)
Proteins deleterious on overexpression are associated with high intrinsic disorder, specific interaction domains, and low abundance.